KNOW Homoeopathy Journal
Volume-1 | Issue-1 | October-2021
Title: Allergic Rhinitis & It’s Homoeopathic Approach
Authored By:- Dr. Ruchi Sharma
PGR-Department of Pharmacy, Swasthya Kalyan Homoeopathic Medical
College and Research Centre, Sitapura, Jaipur, Rajasthan, India.
Cite this Article as:
Dr. Ruchi Sharma, Allergic Rhinitis & It’s Homoeopathic Approach, Vol.1 & Issue 1, KNOW Homoeopathy Journal, Pages 45 to 59 ( 19 October 2021), available at https://www.knowhomoeopathyjournal.com/2021/10/allergic-rhinitis-its-homoeopathic-approach.html
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KNOW Homoeopathy Journal
Volume-1 | Issue-1 | October-2021
ABSTRACT: Allergic
rhinitis is an inflammation of the nasal mucosa induced by allergens and
IgE-mediated inflammation. Allergic rhinitis symptoms include rhinorrhea,
sneezing, nasal irritation, and nasal congestion. Asthma, atopic dermatitis,
and nasal polyps are among the disorders connected to it. Around 20-30% of
Indians suffer from allergic rhinitis, which should be treated according to
ARIA standards, while asthma should be treated according to GINA guidelines.
Allergen avoidance, medication, and allergen immunotherapy should all be used
to treat allergic rhinitis. Intranasal corticosteroids are the most effective
therapy for allergic rhinitis, and their secondary qualities are critical for
patient compliance.
KEYWORD: Allergic
rhinitis ,asthma ,nasal polyps, immunotherapy, homoeopathic management
INTRODUCTIONSneezing,
itching, watery nasal discharge, and a sensation of nasal blockage are all
symptoms of allergic rhinitis, an IgE-mediated hypersensitivity illness of the
nasal mucous membranes. It is
caused by pollens, dust, animal dander, molds, and other allergens in the air.
It's a common chronic disease that affects ten percent to thirty percent of
adults and up to forty percent of youngsters throughout the globe. As pollution
in the environment continues to grow, allergic rhinitis has become a worldwide
health issue. There
are two sorts of it: seasonal and perennial. Allergic rhinitis has been
demonstrated to have a deleterious impact on emotional well-being and cognitive
function. Rhinoconjunctivitis is common in underdeveloped nations, with 15.3
percent of 11 to 15-year-old schoolchildren in Northern Africa affected.Rhinitis
has a negative impact on a person's quality of life as well as their ability to
attend school and work. It has a considerable impact on health-care expenses.
Allergies are expected to cost the United Kingdom's national health systems one
billion pounds per year.Furthermore,
research linking rhinitis and asthma is accumulating. Science has improved not
just our knowledge of allergies, but also the results of allergy treatment. If
allergic rhinitis is not properly treated, it may progress to bronchial asthma,
bronchitis, eczema, and other allergy symptoms.Traditional
medicine assumes that all colds are the same and prescribes the same set of
drugs, including intranasal corticosteroids, antihistamines, decongestants,
nasal irrigation, and unnecessary surgery; sometimes to dry the nose, sometimes
to suppress the cough, sometimes to reduce the fever and headache, and all of
these drugs can have side effects.Homoeopathy,
on the other hand, may provide a considerable cure without any side effects or
the need for unneeded surgery by encouraging the body to heal itself and so
healing the patient's problems holistically, since it is a system that focuses
on the individual rather than the illness. The holistic approach is utilized in
homoeopathic treatment, which treats the individual as a whole rather than
focusing on the disease's symptoms.
DEFINITION[1]:”Allergic
rhinitis is an IgE mediated
hypersensitivity disease of the mucous membranes of
the nasal airways
characterized by sneezing, itching
in the nose , watery nasal
discharge and a sensation of
nasal obstruction.”The lining
of the nose
is continuous with
the paranasal sinuses which may
also be involved. Associated allergic
conjunctivitis and bronchial
asthma may occur.Allergic
rhinitis occurs in
atopic individuals who
are exposed to
common aeroallergens.
CLASSIFICATION OF
ALLERGIC RHINITISA. Clinical types[2]:1. Seasonal allergic
rhinitis
- it occurs
particularly during pollen
season to which patient
is sensitive.2. Perennial allergic
rhinitis
-
in this rhinitis
symptoms are present
throughout the year.A. According to
severity[3] :1. Mild –
intermittent2. Moderate- Severe intermittent3. Mild – Persistent4. Moderate – Severe PersistentIntermittent is when
the symptoms occur <4 days per week or <4
consecutive weeks.Persistentis when
symptoms occur >4 days/ week and
>4 consecutive weeks.The symptoms
are considered mild with
normal sleep , no impairement
of daily activities, leisure , no impairement of work
and school, and if
symptoms present but
not troublesome. Moderate –
severe symptoms result in
sleep disturbance ,
impairement of daily
activities, sports , leisure , impairement of
school or work
and troublesome symptoms.
PATHOGENESIS Allergic rhinitis arises when a person with a sensitized immune system inhales an allergen such as pollen, dust, or animal dander (particles of lost skin and hair). In some persons, the allergens induce the antibody immunoglobin E (Ig E) to form, which binds to histamine-containing mast cells and basophils. When it's caused by pollen from any plant, it's termed "pollinosis," and when it's caused simply by grass pollens, it's called "hay fever." Hay fever has nothing to do with fevers or hay, yet it is named from the pollen released into the air by grasses when hay is harvested.[4] Pollen and dust trigger IgE binding to mast cells, resulting in the production of inflammatory mediators such as histamine and other substances. Sneezing, itchy and watery eyes, oedema and inflammation of the nasal passages, and an increase in mucus production are all common symptoms. [4]In reaction to inhaled allergens, genetically predisposed persons produce specific IgE antibodies. The Fc end of this antibody clings to blood basophils and tissue mast cells. Antigens combine with IgE antibody at the Fab end after further exposure. Mast cells degranulate as a consequence of this event, releasing a range of chemical mediators, some of which are already produced and others that must be created from scratch. These mediators are responsible for the symptomatology of allergic disease. Vasodilation, mucosal oedema, eosinophil infiltration, increased secretion from nasal glands, and smooth muscle contraction may occur depending on the tissue involved.[5]Clinically
, allergic response occurs
in 2 phases:a)
Acute or
Early phase –Sneezing, rhinorrhoea, nasal blockage, and/or bronchospasm are common symptoms that appear 5-30 minutes after contact to the allergen. The release of histamine and other vasoactive amines causes it.b)
Late or Delayed
phase – It occurs after an allergen has been exposed for 2–8 hours with no additional exposure. Eosinophils, neutrophils, basophils, monocytes, and CD4 + T lymphocytes invade the antigen deposition site, causing swelling, congestion, and thick secretion. When an allergen is exposed repeatedly or continuously, the acute and late phases of symptoms overlap.[5]
AETIOLOGY
A. Genetic predisposition : If one or both parents have allergic rhinitis, the chances of their offspring acquiring allergies are 20% and 47%, respectively[6]
B. Allergens[7]:
1.
Seasonal allergic
rhinitis – It is
commonly caused by allergy
to seasonal pollens
and outdoor molds.
2.
Grass pollens - seasonal allergic rhinitis in U.K (Varney, 1991) is most commonly due
to allergy to grass
pollen with seasonal
symptoms in june or july
corresponding to peak
grass pollen counts. The commonest grass species
associated with allergic
rhinitis are – perennial rye
(Loliumperenne), the large leafed Timothy grass (phleumpratnese), cocksfoot (Dactylisglomerata), meadow
fescue (Festuca pratensis) , Yorkshire
fog (Holcuslanatus).
3. Tree pollens –
Seasonal
allergic rhinitis occurring during the
spring time may occur
following exposure to
tree pollens including birch, hazel , plane tree, ash
and pine.
4.
Weed pollens -includes nettle , dock ,and mugwort flower
in late summer.
5.
Fungi spores- in late summer and
autumn. Common species includes Cladosporium, Alternaria
, Aspergillus and
Basidiospores.
6.
Paradoxically,
rainfall may provoke
an initial increase
in spore numbers.
7.
Perennial allergic
rhinitis -
worldwide
the commonest cause
of perennial allergic
rhinitis is allergy
to –
House
dust mite species including D.pteronysinnus , D. Farina andEuroglyphusmaynei.
House
dust are
the dominant allergens
in house dust. The
optimalconditions for
mite growth are
approximately 15-20 degree
Celsius, relative humidity
60-70%. Mites are more
abundant in humid
homes. In houses the
bedroom is the
preferential breeding ground, particularly the
mattress where abundant
food, in the form
of human skin
scales, is present.Mites also
flourish in the
pillow, bed clothes, carpets,
curtains and soft furnishings.The major
allergens of house
dust mite have
been identified as
digestive enzymes ( cysteine proteases, group 1 allergens)
present in the digestive tract
and excreted in
mite faeces.
Domestic
pets –
allergy
to pests is
also a common
cause of perennial
allergic rhinitis- cats, dogs, rabbits, guinea pigs,
gerbils,hamsters , horses and cockroach.
1. Occupational allergens - Rhinitis may
occur as a consequence
of exposure to
allergens inhaled in
the workplace. Frequently,
asthma and rhinitis
due to occupational
allergen exposure may
coexist in the
same patient.
Common biological
causes include –
flour (in bakers, grain workers), laboratory animals including guinea pigs, rats and
mice (in lab. Workers) and wood dusts, biological washingpowders( in soap
powder manufacturers) and colophony
(due to the emanations
of soldier flux in electronic workers).
Latex allergy –
Recently
identified allergen is
latex. It may provoke rhinitis, asthma, urticaria and,
occasionally, life threatening anaphylaxis. Susceptible workers
include surgeons , nurses, dental
nurses, other health workers and
patients with indwelling
latex urinary catheters (eg. Spina bifida
patients). Other chemicals
causes include platinum
salts and drugs.
A. Food induced
rhinitis : food may
occasionally provoke IgE-
mediated allergic rhinitis.In
addition to IgE-mediated mechanism, food induced
rhinitis may be due
to sensitivity to preservatives such
as sulphites, benzoates and tartrazine.Histamine- containing
foods
such as cheese, poorlykept fish
and certainwinesmay also
provoke ‘pseudo – allergic’
reactions including flushing, headache and
rhinitis.Alcohol may
provoke nasal congestion.
2.
Food induced
allergic rhinitis is more common
in children.
3. Drug induced
rhinitis :the mechanism
of drug- induced rhinitis
is largely unknown. Following drugs
can cause allergic
rhinitis:
Sensitivity to
aspirin , NSAID’s , antihypertensive
drugs, topical vasoconstrictors.
1.
Role of
pollution :Nasal hyper- reactivity refers
to heightened sensitivity
of the nasal
mucosa to a
range of non-specific
irritants. Typical irritants include
perfumes, domestic sprays,
tobacco smoke, traffic fumes and
bleach.
CLINICAL
FEATURES
There is
no age or sex predilection. It may
start in infants
as young as 6 months
or older people. Usually the
onset is at 12-16 years of age.Effective treatment
of rhinitis symptoms
depends upon accurate
clinical diagnosis and
assessment of the
patient’s dominant symptoms. In
general, the diagnosis of
allergic rhinitis is
straightforward and dependent
upon the clinical
history. However, the aetiology of
rhinitis symptoms is
frequently multifactorial.A careful
history, local examination of the nose
and performance of
skin-prick tests should
be performed in
all patients presenting
with rhinitis symptoms. Additional tests
including flexible and
rigid endoscopy, mucociliary clearance
studies and immunological
tests may be
required in certain
circumstances.
SYMPTOMS[8]
A.
Seasonal Rhinitis
1.
Sneezing - The firstsymptom
of the hay
fever season is
usually sneezing. In severe
cases paroxysms of
sneezing occur at
frequent intervals throughout
the day. Sneezing is
probably largely due
to histamine release
acting through reflexes.
2.
Rhinorrhoea- Excessive fluid
and mucous secretion
is believed to
be the response
of seromucus glands
to mast cell/ basophil derived
mediators.
3.
Nasal obstruction or blockage –is the
result of vascular
engorgement with resulting
vasodilatation and oedema
formation.
4.
Itching of
nose, eyes, and palate – are common
features resulting from
histamine and/or neural reflexes.
5.
Tearing
, itching and redness of
the eyes- together with
some degree of
periorbital oedema is usual
in hay fever.
6.
Other symptoms- may include
a burning or raw sensation in
the throat and
development of asthma
symptoms such as
wheezing and chest
tightness.
B
.Perennial Rhinitis
1.
The symptoms
of perennial rhinitis
differ from seasonal
rhinitis largely as
a result of
long-standing nasal mucosal inflammation.
2.
Rhinorrhoea
- may be
more viscous or
purulent depending on
the degree of
cellular recruitment.
3.
Conjunctivitis-
is
far less frequent
in perennial rhinitis.
4.
Loss of
smell, taste and associated sinusitis or
Eustachian tube dysfunction.
5.
In general, sneezing- is less
common and prolonged
continuous symptoms of
nasal congestion and
postnasal drip are
more common.
SIGNS[9]
A. Nasal signs -includes
1. Transverse nasal
crease - a black
line across the
middle of dorsum
of nose due
to constant upward
rubbing of nose
simulating a salute (allergic
salute).
2. Pale and
oedematous nasal mucosa
which may appear
bluish.
3. Turbinates are
swollen.
B. Ocular
signs – includes
1.
Oedema of lids,
congestion and cobble-stone
appearance of conjunctiva.
2.
Dark circles
under the eyes (allergic
shiners)
3.
Increased lachrymation
with long and
silky eyelashes.
4.
Dennie- Moragan lines
(creases in the
lower eyelid skin) caused by venous
stasis may be
present.
C. Otologic signs
– includes
1. Retracted tympanic
membrane or serous
otitis media as
a result of
Eustachian tube blockage.
2. Eczematous otitis
externa may be
present.
D. Pharangeal signs - include
granular pharyngitis due
to hyperplasia of
sub mucosal lymphoid
tissue, which are also
termed as “ cobblestoning”
E. Laryngeal signs - include hoarseness
of voice and
oedema of the
vocal cords.
F. Skin –
Atopic
dermatitis may be
sometimes found with
allergic rhinitis.
G.
Mouth
– Mal-
occlusion (overbite) A high arched
palate, narrow premaxilla and
receding chin may
be present secondary
to excessive mouth
breathing.
DIAGNOSIS OF
RHINITIS[13]
Obtaining a full history and completing a physical examination accompanied with crucial tests are used to diagnose rhinitis in a patient who is complaining of upper airway issues. Additional laboratory, radiologic, and morphologic tests may be conducted if they are deemed essential.
To separate rhinitis from upper respiratory infections or other nasal problems, a complete history supplemented with particular questions, delivered in the form of either a structured oral interview or a written questionnaire, is required. The following items should be included in such a questionnaire: -
Is there a history of atopy in your family?
Is there a dominant nasal symptom, such as obstruction, sneezing, or nasal discharges, in the symptom profile?
Are the nose issues isolated or do you have other symptoms as well?
Are there any signals from other sections of the upper airways,that you should be aware of?
Is there a history of bronchitis, eye disease, or dermatologic disease?
What would you use to describe the symptoms, and what is the timeline for their onset?
Are there any allergens in the house environment, such as bedding materials, pets?
Are there any specific precipitating causes (pollen, for example)?
Is there a connection between food and drink? Do any fresh fruits or veggies irritate your mouth?
What medications are you now taking? Does any medication make symptoms worse?
What are your job and leisure activities, especially those that worsen your symptoms?
Symptoms
Nasal obstruction, itching, sneezing fits, and increased nasal surface fluid are the classic symptoms, however the primary symptom varies from patient to patient. There is also a lot of diversity in how well nasal symptoms are tolerated by different people. Some individuals find a few episodes of sneezing bothersome, while others ignore the fact that their nasal canal is fully closed and seek medical help. When it comes to determining the severity of rhinitis, a complete symptom score registration may be beneficial.
Pathogenesis difference leads to variability of symptoms of the major nasal symptoms. It is the result of a decrease in the tone of the capacitance vessels and, to a minor degree, tissue oedema. The increase in nasal surface liquid is the result of glandular activity, the leakage of plasma, and the increase in fluids from other sources, such as the conjunctiva.
Conjunctival
symptoms of itching
and increase in
tear fluid are also very
common in association
with allergic rhinitis: the
term rhinoconjunctivitis is
often more relevant.
Physical Examination
§
Several facial
features are associated
with the various
symptoms of the
nasal and ocular
disease. These include:
·
“Allergic shiners” - infra orbital
dark circles, related to
venus plexus engorgement.
·
“Allergic gape “
or continuous open-
mouth breathing – a result
of nasal blockage.
·
“Transversal nasal
crease” – a
result of frequent
upward rubbing of
the nose ‘allergic salute’ ; and
·
“Dental malocclusion and overbite”
resulting from
long standing upper
airway problems.
D.Rhinoscopy
The
following findings should
be noted:
o Any
structural deformities, such as
septal deviations- site of
any deformity should
be specified and
the presence or absence of
polyps should be
recorded.
o The
amount and the
condition of nasal
surface liquids (e.g. watery ,
mucoid , or purulent)
o The
condition of mucous
membranes and the
colour , texture, and signs of
scars and lesions
should be specifically
evaluated – an allergic condition
might be indicated
by the traditional
bluish tint
o Unilateral
nasal obstruction may
also indicate a foreign
body.
E.Examination
of extranasal regions
The eyes, ears, chest, and skin are some of the other areas that should be examined.
Patients with persistent rhinitis should have their medical history collected to rule out asthma, their chest inspected, and some type of lower respiratory tract functional measurement, such as peak flow or spirometry, performed.
Because otitis media and middle ear effusions might occur more frequently in children with allergic rhinitis, the ears should be examined thoroughly, with a focus on any middle ear pathology. The otomicroscope is the ideal tool for this. A tympanometric examination can also be beneficial. Atopic skin illnesses may manifest themselves more frequently, so the doctor should check for urticaria or eczematous lesions.
Atopic skin
diseases also
may occur with
increased frequency, so
the physician should
check for urticaria
or eczematous lesions.
D. Additional
tests
1. Tests for
the presence of
allergy-
a.
Skin
tests- help to
identify specific allergen. They areprick, scratch and
intra- dermal tests.
Skin tests are preferred
to scratch or
intra- dermal tests which
are less reproducible, more dangerous
and may give
false-positive response.
b. Blood tests-
·
Total and
differential count- to
determine blood eosinophils.
High level indicate Atopy (predisposition to develop
allergic disorders ).
·
RAST
(Radio allegro-sorbent test) - is
an in-vitro test
and indicates the
presence of a specific
IgE to any
of the more common airborn
allergens.
·
Serum
IgE level- high
level indicates atopy.
2. Nasal
challenge-
Nasal
challenge can be
used to test
for specific as well
non-specific reactivity. Non-specific
reactivity may be
tested using methacholine
and histamine as
the challenge agents. The
test for specific
reactivity involves the
application of the
specific allergens to
the nasal mucosa.
3. Cytologic studies-
To assess the
severity of the
disease, or to elucidate
whether the upper
airway disease is
of allergic origin, examination of the cytology
of the upper
airway mucosa is
required. Techniques arenasal smear
and biopsy.
The main focus
is on to
demonstrate the presence
or absence of
eosinophils. The presence of eosinophils is
a sign of
active inflammatory disease
of allergic origin.
1. Radiology-
Radiographic
studies needed to
establish the evaluating
possible structural abnormalities
or to help to
detect complications or
co-morbid conditions such
as sinusitis or
adenoid hypertrophy.
a. A plain
radiography of the
sinus region.
b. A lateral
view of neck.
c. Computed
tomography( CT) scan.
COMPLICATIONS OF
ALLERGIC RHINITIS[10]
1.
Bronchial asthma
2.
Recurrent sinusitis
3.
Serous otitis media
4.
Nasal polypi
TREATMENT
OF ALLERGIC RHINITIS[11]
1.
Avoidance of
allergens: this is
most successful if the antigen
involved single. Removal
of pet from
the house, encasing the
pillow or mattress
with plastic sheets, change of
place of work
for some times change
of job may
be required. A
particular food article
to which the
patient is found
allergic can be
eliminated from the
diet.
2. Treatment with
drugs :
a. Antihistaminics
b. Sympathomimetic drugs (oral
or tropical)
c. Corticosteroids
a.
Sodium chromoglycate
HOMOEOPATHIC APPROACH
Dr. Samuel Hahnemann was the first person to introduce Homoeopathy to the world of medicine.
The Law of Similars, which has been recognized since Hippocrates' time and is also noted in ancient Hindu literature, is the foundation of homoeopathy. However, it took more than 200 years for Dr. Samuel Hahnemann, a German physician, to completely grasp, implement, and integrate the Law of Similars into a comprehensive therapeutic system.
In his book "The Organon of Medicine," Hahnemann discusses the principles of Homoeopathy, which include the spiritual genesis of illness and the medications needed to heal it. Organon's Aphorisms 9 and 16 in particular deal with this subject. Homoeopathy is a holistic healing technique that blends science and spirituality.
Allergic
rhinitis is one
of the disease
that can be
cured completely and
permanently only with
the help of
Homoeopathy.There is special
mention about allergic
diseases in homoeopathic
literatures under the
heading IDIOSYNCRASY. Dr.Hahnemann
defined idiosyncrasy in
his famous book
Organon of Medicine
as “Idiosyncrasies by
which are meant
peculiar corporeal constitutions
which although otherwise
healthy, posses a disposition
to be brought
into a more
or less morbid
state by certain
things which seems
to produce no
impression and no
change in many
other individuals”.
Allergic responses and idiosyncracies have similar manifestations. Both of these reactions are hypersensitive. An idiosyncratic person is hypersensitive to one or a few items and does not need treatment because they are healthy, while allergic illnesses are caused by morbid susceptibility.
Successful homoeopathic prescribing necessitates a thorough case taking to determine the progression of the disease, its relationship to the patient's life condition, and the presence of specific symptoms. According to Hahnemann, an individual's constitution is what causes them to become unwell in the first place. The goal of treatment should be to improve the patient's constitution so that their sensitivity, which has deteriorated, can be repaired or brought back into normal range. It has been proven, verified, and confirmed that providing a child with appropriate constitutional treatment for a period of time enhances the child's constitution. As time passes, the attacks grow less frequent and less severe. By using a constitutional strategy, we can prevent the complaint from reoccurring by altering the changed susceptibility.
Miasmatic classification of
Allergic Rhinitis[13]:-
· A hypersensitive sensitivity to different things such as dust, particular meals, or even strain causes a Psoric recurrent cold. Sneezing, a runny nose, and fatigue, with or without a fever, are all symptoms. It gets back on its feet without effort. The cough will be dry and spasmodic, with minimal expectoration, and it will become better with rest and heat.
·
In the long term, the Sycotic kind of reoccurring cold may lead to chest congestion and asthma. There may be a loss of smell, sneezes in youngsters, and a dry rattling cough that occurs after a brief exposure to the cold. The process of recuperation is gradual.
· The tubercular kind of recurrent cold will result in repeated infections of the tonsils and lymph nodes. A deep cough with purulent greenish yellow expectoration, post-nasal drip, thick catarrhal discharge, haemorrhages, and a post-nasal drip are all potential signs. Overheating and nighttime (sunset to dawn) aggravate the condition, while nose bleeds and cold application relieve it. As a consequence of this, there will be prostration. Here, recurrence will be frequent, and recovery will be partial.
·
It can cause ulcerations of the nasal septum, glands, and other tissues during the Syphilitic stage. It will be difficult to recover.
HOMOEOPATHIC THERAPEUTICS
OF ALLERGIC RHINITIS[12][14]
1.)
ALLIUM CEPA : "Coryza copious,
watery, and acrid with profuse, bland lachrymation", worse in a hot room
and later in the evening, better in the open air. Sneezing, particularly when
entering a hot environment.
2.) AMBROSIA : "Lachrymation and
incessant itching of the eyes." Watery coryza, sneezing, and epistaxis. In
the nose and head, there is a stuffy sensation.
3.)
ARALIA RACEMOSA: Sneezing
on a regular basis. Draughts irritate greatly. Sneezing and abundant watery,
excoriating nasal discharge with a salty bitter flavor are caused by the least
circulation of air.
4.)
ARSENIC ALBUM : "There is a lot of
restlessness, and pain moves about a lot." Fear of dying and being left
alone. "Extreme weariness after the tiniest effort." Acrid
lachrymation and a burning discomfort in the eyes. Nasal discharge that is
thin, watery, and excoriating. The bridge of my nose had become clogged.
Sneezing and sneezing and sneezing and patient has a lot of thirst and drinks a
lot of water, but just a little at a time. Worse in the middle of the day and
at night; better with warmth and warm liquids.
5.)
SABADILLA: Chilliness, cold sensitivity Lachrymation has
made my eyelids red and burning. With a runny nose and spasmodic sneezing.
Dryness of the mouth and throat. Warm food may be swallowed more readily.
6.) HEPAR SULPHUR: Physically and intellectually, patient is very
sensitive to all sensations. Even in warmer weather, he must have his face bundled
up since he is very sensitive to chilly air. Nasal congestion caused by
catarrh. When exposed to a cold, dry breeze, he sneezes. A splinter sensation
in the throat. Suppuration may result from very minor injuries.
7.)
EUPHRASIA: Acrid lachrymation and bland coryza in
abundance. Eyes are constantly watering and agglutinated in the morning. Coryza
in the morning with a strong cough and a lot of expectoration, aggravated by
exposure to the south wind in the evening.
8.) LEMNA MINOR: Polyps in the nose;
enlarged turbinates. Atrophic rhinitis is a kind of atrophic rhinitis. Nasal
blockage causes asthma. Nose emits a foul odor. There are a lot of crusts and
mucopurulent discharge. Nasal leaking thereafter. When you wake up in the
morning, you have a sour taste in your mouth. It's much worse when it's raining
heavily and it's moist.
9.) KALI
BICHROMICUM: "Tough,
stringy mucus that sticks to the parts and may be dragged into long
threads" is discharged from all mucous membranes.[14] Pain in
little places, which may be covered by the tip of the finger, and which shifts
fast. Pain at the root of the nose; plugs, or "clinkers," are
discharged. Coryza accompanied with a nasal blockage. Sneezing with venom.
Oedematous uvula, bladder-like. When it's hot outside, it's much worse.
10.) SAMBUCUS NIGRA : Infants with dry coryza sneeze; nose is
dry and clogged. When a kid is breastfeeding, he or she must let go of the
nipples, which causes the nose to get blocked and the youngster to be unable to
breathe. "The child wakes up, almost suffocating, stands up, and becomes
blue." It cannot be said to have expired. "During awake hours, sweat
profusely all over body."
References
1. Scott-Brown’s Otolaryngology- 6th edition, volume 4- Rhinology , Chapter 6,
Page- 4/6/1
2. Scott-Brown’s Otolaryngology- 6th edition, volume 4- Rhinology , Chapter 6,
Page- 4/6/1
3. Allergic
rhinitis and its impact on asthma –
ARIA, 2007, page no. 4
4. Allergic rhinitis and its impact on
asthma- ARIA, 2007,page no. 2
5. P.L Dhingra Diseases
of Ear, Nose and
Throat, 5th edition
Chapter 30 , page 180 – 181
6. P.L Dhingra Diseases of
Ear, Nose, Throat, 5th edition, chapter 30, page 180
7. Scott- Brown’s Otolaryngology- 6th edition, volume 4- Rhinology, Chapter 6, Page
-4/6/2 to 4/6/4
8. Scott- Brown’s Otolaryngology- 6th edition, volume 4- Rhinology, Chapter 6, Page
-4/6/8
9. P.L Dhingra Diseases of
Ear, Nose and Throat,5th
edition Chapter 30,page 181
10.Scott- Brown’s Otolaryngology- 6th edition, volume 4- Rhinology, Chapter 6, Page
-4/6/9-4/6/10
11.P.L.Dhingra diseases of
Ear, Nose and Throat, 5th edition , Chapter 30, page 182
12.Boericke
W. , Pocket Manual of
Homoeopathic Materia Medica
13.NASO RESPIRATORY
ALLERGIES AND HOMOEOPATHY – Dr. Prasanna .... https://doctorprasanna.com/articles/naso-respiratory-allergies-and-homoeopathy/
14.H.C. Allen, Allen's Keynotes and Characteristics with Comparisons
KNOW Homoeopathy Journal
Volume-1 | Issue-1 | October-2021
Cite this Article as:
Dr. Ruchi Sharma, Allergic Rhinitis & It’s Homoeopathic Approach, Vol.1 & Issue 1, KNOW Homoeopathy Journal, Pages 45 to 59 ( 19 October 2021), available at https://www.knowhomoeopathyjournal.com/2021/10/allergic-rhinitis-its-homoeopathic-approach.html
